Prions and Neurodegenerative Disease Risk

I dont need a whole paper:: all i need to add ONE page of data/research and/or a visual to this paper supporting the hypothesis. Please note the data should be a form of a graph, list or chart. The data analysis not only supports the conclusion but also explains addi‌‍‍‌‌‍‌‌‌‌‌‍‍‍‌‌‌‌‍tional relationships. Patterns and relationships in the data are identified are identified and fully explained. Additional relationships may be predicted. Any Visuals or sources used need to be listed in the references in the APA format. Paper is Uploaded as a file‌‍‍‌‌‍‌‌‌‌‌‍‍‍‌‌‌‌‍.

Mutation	Onset	Familial history	Clinical Phenotype	Region reported
Octapeptide insertions	Frequently early onset	Frequently familial	At most, nine octapeptide insertions. CJD-like phenotype amyloid plaques might be dominant. Highly transmissible	Asia, Europe
Dual deletion of Octapeptides	indeterminate	indeterminate	CJD and swiftly increasing dementia.	USA
M232R	Fluctuates	Infrequent and familial	Swift, Usual CJD, Slow continuous CJD with dementia, DLB-related symptoms	China, Japan, Korea
I215V	The seventies or fifties	De novo	CJD or AD	Spain
V2101	Fluctuates	Familial or de novo	Usual CJD, infrequent CJD	Europe, Africa, Asia
R208H	Fluctuates, 45-69 years	Infrequent	Anorexia, ataxia, CJD, cognitive decline agitation	China, Europe
V203I	The seventies and eighties	Infrequent mostly	Hallucinations, Dizziness, Monocular Diplopia, Tremor, coordination deficit, myoclonus, rapid vocabulary, and memory loss	France, Korea, Japan, China
E200G	NA	Infrequent	CJD	UK
E200k	Between the thirties and sixties	Familial and infrequent	CJD spongiform degeneration, dysfunctional spinal cord, and central nervous system	Jewish Libyans Europe Asia

This question has been solved!

Verified by Admin
Written by a Human Expert

Unlock Solution Get Custom Solution

Question

Prions and Neurodegenerative Disease Risk

Expert Solution

This question has been solved!

Related Questions