Both developmental prosopagnosia (DP) and acquired prosopagnosia (AP) are characterised by a deficit in recognising faces. Are the causes and mechanisms the same in DP and AP? use neuropsychological literature to support answer
The capacity to identify known faces, including one's own, is diminished
in prosopagnosia, a cognitive disease of face perception, although other
elements of optical and perceptual functioning remain unaffected. The word was
first used to describe a condition that followed acquired prosopagnosia, but
there is also a developmental version of the illness with an incidence of less
than 3% (Albonico
& Barton, 2019). The two forms of prosopagnosia share various
characteristics but are intrinsically distinguished by various causes and
mechanisms associated with their development in individuals. Prosopagnosia is
often associated with the fusiform gyrus, a brain region that predominantly
reacts to faces. Due to the working of the fusiform gyrus, most people can
distinguish faces more accurately than comparable complex inanimate objects
(Albonico & Barton, 2019). The simpler object-recognition system is what
allows prosopagnosics to recognize faces. Furthermore, recognized face pictures
are more commonly associated with the right side of the brain's fusiform gyrus
than the left. But it is still uncertain whether the fusiform gyrus
participates in highly refined sensory input or merely in recognition of
individual faces (Adams et al., 2019). Thus, prosopagnosia is categorized as
acquired or developmental, depending on various mechanisms and causes.